By Jane Williams, Lin Perry, Caroline Watkins
Chapter 1 atmosphere the Scene (pages 1–16): Professor Caroline Watkins and Michael Leathley
Chapter 2 constructing Stroke prone: A Key position for Nursing and Nurses (pages 17–32): Christopher R. Burton
Chapter three what's a Stroke? (pages 33–65): Anne W. Alexandrov
Chapter four Acute Stroke Nursing administration (pages 66–90): Anne W. Alexandrov
Chapter five dietary features of Stroke Care (pages 91–122): Professor Lin Perry and Elizabeth Boaden
Chapter 6 selling Continence (pages 123–151): Kathryn Getliffe and Wendy Brooks
Chapter 7 administration of actual Impairments Post?Stroke (pages 152–183): Cherry Kilbride and Rosie Kneafsey
Chapter eight communique (pages 184–204): Jane Marshall, Katerina Hilari and Madeline Cruice
Chapter nine temper and Behavioural adjustments (pages 205–221): Peter Knapp
Chapter 10 Minimally Responsive Stroke sufferers (pages 222–240): Elaine Pierce and Aeron Ginnelly
Chapter eleven Rehabilitation and restoration techniques (pages 241–262): Dr Jane Williams and Julie Pryor
Chapter 12 Stroke and Palliative Care: a tough mix? (pages 263–274): Christopher R. Burton and Sheila Payne
Chapter thirteen lowering the danger of Stroke (pages 275–308): Peter Humphrey, Jo Gibson and Stephanie Jones
Chapter 14 Longer?Term aid for Survivors and Supporters (pages 309–330): Louise Brereton and Jill Manthorpe
Chapter 15 Stroke assets for execs, sufferers and Carers (pages 331–341): Graham Williamson
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Additional resources for Acute Stroke Nursing
Reproduced with permission of Stephen DiBiase Designs. tricles extend from the frontal to the occipital lobes (Standring 2004; Waxman 2000). The right lateral ventricle is commonly the site for cannulation when CSF drainage by ventriculostomy or shunt, and/or intracranial pressure monitoring is required. The two lateral ventricles are connected by the foramen of Monro to the third ventricle, which lies directly above the midbrain of the brainstem. The aqueduct of Sylvius, or cerebral aqueduct, connects the third with the fourth ventricle, which is located between the brainstem and the cerebellum.
Patients with this stroke mechanism often demonstrate evidence of widespread atherosclerotic disease, such as intermittent claudication, coronary artery disease, extracranial carotid stenosis and/or TIA occurring in the same vascular territory. For this categorisation, there should not be any indication of a cardioembolic mechanism (Adams et al. 1993). In the Stroke Data Bank of the US National Institute of Neurological Disorders and Stroke (NINDS), large artery atherosclerosis was responsible for about 6% of strokes, with another 4% categorised as tandem artery occlusions (Foulkes et al.
Many clinicians prefer to talk in terms of acute stroke syndrome, rather than differentiate between TIA and stroke, and UK guidelines consider that using ‘brain attack’ to describe any neurovascular event, ‘may be a clearer and less ambiguous term to use’ (National Collaborating Centre for Chronic Conditions 2008). When blood flow to an area is disrupted through occlusion of blood vessels, two major zones of injury develop: the core ischaemic zone and the ‘ischaemic penumbra’. In the core zone, very low blood flow means oxygen and glucose supply is inadequate and stores are rapidly depleted.